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Peter H. Duesberg1,*, Daniele Mandrioli1, Amanda McCormack1, Joshua M. Nicholson2, David Rasnick3, Christian Fiala4, Claus Koehnlein5, Henry H. Bauer2 and Marco Ruggiero6

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namepdijae.pdf

Author

  • Peter Duesberg

  • Daniele Mandrioli

  • Amanda McCormack

  • Joshua M. Nicholson

  • David Rasnick

  • Christian Fiala

  • Henry Bauer

  • Marco Ruggiero

Publisher

  • IJAE

Topic

Publish Year

  • 2011

Content Type

  • Scientific Paper

Description

Meta Tag

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Summary

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Table 1 – Population statistics of South Africa from 1980 until 2008.

Year  Population

HIV+

HIV-Death

x10-3 (a)

% (b)

x10-3 (c)

1980

29,300

1981

30,200

1982

31,100

1983

32,100

1984

33,200

1985

34,300

1986

35,100

1987

35,900

1988

36,800

1989

37,600

1990

38,500

0.7

1991

39,300

1.7

1992

40,100

2.2

1993

40,900

4.0

1994

41,600

7.6

1995

42,200

10.4

1996

42,800

14.4

1997

43,300

17.0

*

1998

43,900

22.8

*

1999

44,500

22.4

10.0

2000

45,100

24.5

10.5

2001

45,600

24.8

*

2002

46,100

26.5

*

2003

46,600

27.9

*

2004

47,000

29.5

13.0

2005

47,500

30.2

14.5

2006

47,900

29.1

2007

48,400

28.0

2008

48,800

(a)Statistics South Africa and US Census Bureau (Statistics South Africa, 2007; Statistics South Africa, 2000; US Census Bureau, International Data Base, 2008).

(b)National Department of Health South Africa (National Department of Health South Africa, 2007).

(c)  Statistics South Africa (Statistics South Africa, 2007; Statistics South Africa, 2008) Statistics South Africa. Mor- tality and causes of death in South Africa, 2006: Findings from death notification. Statistics South Africa, 2008.

*Not reported because HIV-deaths were below 10th rank.

Moreover, a new viral epidemic causing steady losses of 300,000 per year for 6 years is not compatible with the classic germ theory of disease. Instead, the germ theory predicts that new viruses and microbes cause epidemics that rise exponentially, because of exponential growth and spread of microbes, and then fall exponentially, because of the resulting immunity and deaths within several months, rather than go steady over 6 years (see Fig. 1 and Introduction). HIV has been demonstrated 20 years
ago to induce anti-viral immunity - but not AIDS - within several weeks after infection (Clark et al, 1991; Daar et al, 1991), just like any other virus (Duesberg, 1989). Thus a new virus could have been a plausible explanation for a seasonal epidemic of several months within a given year, but not for a steady loss of lives for 6 years in a row.

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Year

Population x 10-3 (a)

1980

12,400

1981

12,700

1982

13,100

1983

13,500

1984

13,900

1985

14,400

1986

14,900

1987

15,600

1988

16,200

1989

16,800

1990

17,500

1991

18,100

1992

18,700

1993

19,400

1994

20,100

1995

20,700

1996

21,200

1997

21,900

1998

22,500

1999

23,200

2000

24,000

2001

24,700

2002

25,500

2003

26,300

2004

27,200

2005

28,200

2006

29,200

2007

30,300

2008

31,400

a) US Census Bureau (US Census Bureau, International Data Base, 2008).

For this purpose we plotted the HIV-antibody prevalence of the South African popula- tion reported by the National Department of Health South Africa since 1990 (Department of Health South Africa, 2007) on a separate panel of Fig. 4, which shows the population growth curve of South Africa. Fig. 4B shows that anti-HIV antibodies were first detected in 1990 in 0.7% of the population. This percentage then increased gradually (not exponentially!) over about 10 years until 2000, when it leveled off between 25 and 30%.

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