By A. Hässig, H. Kremer, S. Lanka, W-X Liang, K. Stampfli
May 1998
http://www.virusmyth.com/aids/hiv/ah15years.htm
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The continuous failure in the prevention and treatment of AIDS is rooted in the misinterpretation of an inflammatory auto immune autoimmune process as a lethal, viral venereal disease
Summary - The question of the specificity of the anti-HIV antibody test has to be re-evaluated as it was shown that the viral enrichment obtained from co-cultivations of patients lymphocytes with fetal cord blood by BARRÉ-SINOUSSI et al. and leukaemia cells by GALLO et al., exclusively consisted of proteins of the cell types used in the cell culture. This precludes a clear separation of presumed retroviral and cellular proteins or extracellular matrix proteins. In this context it was shown that the anti-HIV antibody test detects autoimmune antibodies directed against cyto-skeletal proteins e.g. the liver cells. Strongly augmented anti-actin autoantibodies is considered close to pathognomonic for chronically active hepatitis. The original assumption that "reverse transcription" from RNA to DNA is evidence for the existence of retroviruses, was wrong. In fact, "reverse transcription" is a vital mechanism for the maintenance the genome. The decrease in numbers of circulating CD4 lymphocytes can be explained by a stress-induced hyper-cortisolism. Up to date, direct HIV-mediated destruction of CD4 lymphocytes could not be proved. The same is true for measuring of the "viral load". Shortcomings of the applied method to quantify the "viral load" do not permit definitive conclusions. Possibly, it may be taken as an expression of a stress-induced weakening of the cellular immune reactions, in the course of which the nucleoside fragments resulting from the current cell turnover are inadequately eliminated. Furthermore, the treatment of patients with nucleoside analogues has a toxic effect on both the genome of the cell-nucleus and the mitochondria. The latter, therefore, may produce insufficient amounts of ATP, causing organ failure and, eventually, death. The synthetic protease inhibitors used these days are associated with serious side-effects. Therefore, it seems worthwhile, in these patients, to bring back the catabolic situation due to whole body inflammation to homeostasis by administering anabolic phyto-polyphenolic compounds.
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